31 July 2004

Aspartame: Avoid It Like the Plague

A friend with bipolar disease recently had severe mood swings. Eliminating the borna virus had helped previously and no borna virus was present, although there were a number of antibiotic resistant bacterial infections in the brain. His physician put her finger on what appeared to be the major contributory cause, half a dozen or more diet drinks a day.

Aspartame is well known to be a potent neurotoxin and recent studies show it aggravates your weight condition by limiting the ability of your body to monitor calories. One of my family members was diagnosed with multiple sclerosis while drinking large amounts of diet drinks. The same tests that produced the diagnosis showed she no longer had the disease after she stopped. The number of FDA complaints about aspartame are thousands of times the number of complaints about ephedra (maybe millions of times). Don't drink this stuff!

BITTERSWEET: Is this the beginning of the end for aspartame?

Is the writing finally on the wall for aspartame, the artificial sweetener used in diet and sugar-free drinks and foods? There's been a very effective campaign to keep the lid on a devastating catalogue of diseases linked to the sweetener, but in recent weeks we've noticed a few straws in the wind.

Some brave souls have put together a film, Sweet Misery, which documents "one of the most pervasive, insidious forms of corporate negligence in history", as a spokesman has put it.

And now academics are getting in on the act, and have shown how aspartame can lead to overeating. A study by Purdue University found that artificial sweeteners disturb our natural abilities to monitor calorie intake.

Not that any of this is revelatory. Government agencies have known for decades that aspartame is deadly. It was once on the Pentagon list of bio-warfare chemicals submitted to Congress and, in 1984, Dr Woodrow C Monte observed: "Methanol (one of the breakdown products of aspartame) has no therapeutic properties and is considered only as a toxicant. The ingestion of two teaspoons is considered lethal in humans." But his warning came too late. Aspartame had been approved in the States two years earlier as a safe food additive.

It took centre stage once saccharin was discredited after studies showed a link between it and bladder cancer. Aspartame was seen as a good substitute - and one that packed a kick for all those with a sweet tooth, as it is 200 times sweeter than sugar. Its rise continues unchecked today, especially as its patent has recently expired. Around 5,000 products on the market contain the sweetener, and the list is growing by the day, and includes diet sodas, fruit drinks, frozen lollies, instant breakfasts, chewing gum, cocoa and other instant drinks, supplements, drugs, and yoghurt.

Because it is a food additive, no post-marketing trials of its safety are needed. It's not even listed as an ingredient on some products such as vitamin supplements. Even so, aspartame accounts for more than 75 per cent of adverse reactions to food reported every year to the Food and Drug Administration (FDA). Some of these reactions are very serious, including death.

Despite these alarm calls, it's been down to individual scientists to investigate the dangers of aspartame. Aspartame is a dipeptide (that started life as a drug for peptic ulcer). It's made up of L-phenylanine (50 per cent), aspartic acid (40 per cent) and methanol (10 per cent). Some claim that phenylanine on its is a health hazard, and that certainly becomes more likely if it breaks down to methyl ester which, in turn, becomes methyl alcohol or methanol (remember, two teaspoons is lethal.) If it doesn't kill you, methanol can cause blindness. The US Environmental Protection Agency advises daily maximum methanol consumption of just 7.8 mg - and yet many cans of diet sodas contain twice that, and a diabetic using aspartame all day could consume 30 times that amount.

So what causes this breakdown? High heat and prolonged storage have both been shown to transform aspartame into a more dangerous substance. An interesting study linked Gulf War syndrome to diet soda drinks that were kept out in the hot desert sun.

The evidence is becoming too overwhelming for the authorities to continue to ignore this sweetener. It's now been linked to multiple sclerosis, Parkinson's, migraine and headache, brain cancer, chronic fatigue and epilepsy.

The history of aspartame with citations on publications can be found on holisticmed.com. Here is an excerpt:

The discovery of aspartame is reported in the well-known publication, Science (Cloninger 1970)... G.D. Searle approached Dr. Harry Waisman, Biochemist,Professor of Pediatrics, Director of the University ofWisconsin's Joseph P. Kennedy Jr. Memorial Laboroatory ofMental Retardation Research and a respected expert inphenylalanine toxicity, to conduct a study of the effects of aspartame on primates. The study was initiated on January15, 1970 and was terminated on or about April 25, 1971. Dr.Waisman died unexpectedly in March, 1971.

Seven infant monkeys were given aspartame with milk. One died after 300 days. Five others (out of seven total) had grad mal seizures. The actual results were hidden from theFDA when G.D. Searle submitted its initial applications (Stoddard 1995a, page 6; Merrill 1977; Graves 1984, page S5506 of Congressional Record 1985a; Gross 1976b, page 333 of US Senate 1976b).

30 July 2004

Adult Stem Cells and Organ Restoration

Most people are so embroiled in the embryonic stem cell controversy, they don't realize that adult stem cells can be used to rebuild human tissue. This research should proceed full speed ahead and not be caught up in arguments about fetal tissue. Failure to do so will result in millions of lives lost that could have been saved.

In some cases, adult stem cells have been shown to be more effective that embryonic stem cells. People need to do some reading and get their heads cleared up on this. Every day I talk to someone who is totally confused by political diatribes on the issue. Science is being systematically distorted by political agendas and this leads directly to preventable death and disability.

Adult Stem Cells More Effective Than Embryonic Stem Cells in Blood Formation

Because hematopoietic (blood forming) stem cells (HSCs) can restore and maintain blood formation following transplantation into immune deficient hosts, growth of HSCs in culture is important for many clinical applications. Previously, researchers in Sweden used a gene therapy technique to add a growth gene to embryonic stem cells to get adequate growth in culture. According to the authors, however, “HSCs of early embryonic origin, including those derived from differentiated embryonic stem cells, are inefficient in engrafting adult recipients upon transplantation.” The researchers have now shown that adding the same growth gene, Lhx2, to adult bone marrow stem cells allows unlimited growth of the cells. These adult stem cells efficiently rescued immune-compromised mice and generated all blood cells.

27 July 2004

Cellular Invasion: Pathogen DNA Takes Up Residence in Cells

I've been doing a lot of work lately knocking out cells that have been internally infected with pathogens. They cause chronic problems and never function right. Terminating them with electronic frequencies is the only way to get rid of some persistent problems.

The authors of a current paper found parasite DNA embedded in host cells. A letter to the editor noted that this problem is probably found in many disease entities. It is interesting to see tentative findings in my home laboratory confirmed in academic research centers.

Letter to the Editor:

Re: "Chagas parasite invades genome": I think the authors of this paper have been rather modest and have underplayed the importance of their finding that "Typanosomacruzi kinetoplast DNA [is] found in the genomes of infected patients and animals." It could be because their laboratory is dedicated to work on Chagas Disease, and they did not want to give an impression of straying away from it.

The finding has immediate relevance to many other obligate or facultative intracellular pathogens of humans and animals:
- Mycobacterium tuberculosis (Tuberculosis)
- Mycobacterium leprae (Leprosy)
- Listeria monocytogenes (Listeriosis)
- Salmonella typhi (Typhoid Fever)
- Shigella dysenteriae (Bacillary dysentery)
- Yersinia pestis (Plague)
- Brucella species (Brucellosis)
- Legionella pneumophila (Pneumonia)
- Rickettsiae (Typhus; Rocky Mountain Spotted Fever)
- Chlamydia (Chlamydia; Trachoma)

In my opinion, the top two items in the above list are of great importance, and it might be worthwhile for researchers to carry out studies similar to those of Nitz et al. on the possibility of the DNA of the pathogen "invading" the host genome.

T.S. Raman, retired biochemist
New Delhi, India

Chagas parasite invades genome: Typanosoma cruzi kinetoplast DNA found in the genomes of infected patients and animals
By David Secko, New Scientist, 23 Jul 2004

How infection with Typanosomacruzi—an intracellular parasite that can hide out in the cells of the body—results in the development of chronic Chagas disease has been a mystery. Now a study in the July 23 Cell reports the integration of T.cruzi DNA into the genomes of infected patients, as well as chicken and rabbit animal models, suggesting that horizontal gene transfer may play a role in T.cruzi host–parasite interactions.

T. Cruzi infects some 16 to 18 million people in Latin America, and one third of these infections are estimated to result in chronic Chagas disease, which may not manifest itself until decades after an initial infection. A "major controversy" in the area of chronic Chagas disease research has been whether the presence of the parasite or an autoimmune reaction is its potential cause, said David Campbell, from the University of California, Los Angeles, who was not involved in the Cell study.

26 July 2004

Electronic Medicine and Population Dynamics


Pathogen populations may be stable, oscillating, or chaotic, depending on the value of lambda in the logistic difference equation. See Robert May's comment on limits to growth: what stops cockroaches from taking over the universe?

The human body is a rainforest of microorganisms and anyone trying to diminish the population of pathogens or accelerate the growth of helpful organisms needs to understand the basics of evolutionary biology, the growth of populations, and environmental biology.

In particular, the basic mathematics of population growth must be understood to interpret the results of applying electoronic frequencies to populations of pathogens. A good summary of advances in thinking over the last century can be found on Arcytech's site on Population Growth and Dynamics. In particular, the page on "Interesting Facts about Population Growth Mathematical Models" is worth reading.

You will find reference to Robert May's 1976 paper in Nature (see abstract below) which introduced the concept of chaotic behavior into the mathematics of population growth. Professor May has an interesting summary of his findings in "The Chaotic Rhthyms of Life" where you will find a readable article on the logistic difference equation. A single parameter in this equation describes why electronic frequencies appear to work well sometimes and not well at all on other occasions.

In particular, this single parameter explains why electronic frequencies can help people feel better one day and worse the next. An endless cycle of good and bad days leads many people to conclude that electronic frequencies do not work, or are undependable. Much of this can be explained by the use of the wrong frequencies in trying to eliminate a microorganism. However, even with the right frequencies the power transfer from the electronic frequencies to the organism may be high or low and this directly affects the critical parameter in the logistic difference equation.

Let's review Sutherland's five rights for electronic medicine.  The right power level applied to the right organ system with the right frequencies for the right amount of time using the right protocol will always kill a pathogen.

The power transfer can be such that the lambda parameter in the logistic difference equation is less than one. In that case you will be successful if you apply the frequency for a sufficient length of time. The time required is determined by how close lambda is to one. If power transfer can only bring lamda into the 1-3 range, you will have a stable population of pathogens that you cannot eliminate. If it is above three you will have chaotic behavior and large cycles of population growth and dieoff. This phenomenon is undoubtedly one of the contributors to lack of understanding of the so-called Herxheimer effect.

Power transfer can be significantly enhanced by exact frequencies, plate zapping, and the technology of the device applying frequencies. When you get poor results from electronic frequency devices it is important to get some expert help. Lack of power transfer sufficient to reduce lambda to less than one may explain many of your problems.

Simple mathematical models with very complicated dynamics
May RM
Nature. 1976 Jun 10;261(5560):459-67

First-order difference equations arise in many contexts in the biological, economic and social sciences. Such equations, even though simple and deterministic, can exhibit a surprising array of dynamical behaviour, from stable points, to a bifurcating hiearchy of stable cycles, to apparently random fluctuations. There are consequently many fascinating problems, some concerned with delicate mathematical aspects of the fine structure of the trajectories, and some concerned with the practical implications and applications. This is an interpretive review of them.

Click here for comments or questions

20 July 2004

Rife 2004 Pre-Conference Research Workshop

If you are attending the Rife Conference in Seattle on October 1-3, 2004, you may be interested in expanding your knowledge of how to use various devices and techniques to target pathogens at Dr. Sutherland's pre-conference workshop. 

You do not need to attend the Rife Conference to attend this workshop. We do hope you attend both and hear Dr. Clark and all the great speakers and exhibits. Some of Dr. Sutherland's comments on plate zapping and other techniques will be relevant to conference presentations.
Dr. Sutherland will discuss and demonstrate research findings on how to use the combination of a Cameron Aurameter, an FSCAN2, and an Advanced BioPhoton Analyzer to precisely target and eliminate pathogens and cell types locally and remotely. Theory and application of techniques discussed on his website will be presented in a two hour session.

Disclaimer: This workshop is for research and educational purposes only and is not designed for diagnosis or treatment of any medical condition.

Due to significant demand, two workshops are now scheduled on 1 October. The first will run from 1pm to 2:45 pm and the second from 3pm to 4:45. Location will be at or near the conference hotel with the room to be announced shortly. The last workshop will end in time for the opening reception of the Rife Conference. Please remember that seating is limited and is on a 1st come, 1st serve basis.
Click here to sign up for the Rife 2004 Pre-Conference Workshop

18 July 2004

Fish Oil and Cancer Prevention

Pharmaceutical grade fish oils will provide many of the cancer prevention effects noted below. Certain tumors occur less frequently, neoplastic transformation is suppressed, tumor cell growth is inhibited, and tumor cell death mechanisms are enhanced. After the new Transfer Factor Plus which enhances immune function 4-6 times, the most important supplement to take is pharmaceutical grade fish oil and I recommend Sears Lab OmegaRx. Both of these supplements have dozens of articles in leading medical journals documenting their effects.
The beauty of pharmaceutical grade fish oil is that it has enough concentrated n-3 fatty acids to generate significant positive effects in brain and heart function (in addition to cancer prevention). It just doesn't make sense to be without it, particularly when you have to be careful to avoid eating too much fish because of mercury contamination.
The traditional diet of Greece and cancer
Simopoulos AP
Eur J Cancer Prev. 2004 Jun;13(3):219-30
The term 'Mediterranean diet', implying that all Mediterranean people have the same diet, is a misnomer. The countries around the Mediterranean basin have different diets, religions and cultures. Their diets differ in the amount of total fat, olive oil, type of meat, wine, milk, cheese, fruits and vegetables; and the rates of coronary heart disease and cancer, with the lower death rates and longer life expectancy occurring in Greece. The diet of Crete represents the traditional diet of Greece prior to 1960. Analyses of the dietary pattern of the diet of Crete shows a number of protective substances, such as selenium, glutathione, a balanced ratio of n-6/n-3 essential fatty acids (EFA), high amounts of fibre, antioxidants (especially resveratrol from wine and polyphenols from olive oil), vitamins E and C, some of which have been shown to be associated with lower risk of cancer, including cancer of the breast. Epidemiological studies and animal experiments indicate that n-3 fatty acids exert protective effects against some common cancers, especially cancers of the breast, colon and prostate. Many mechanisms are involved, including suppression of neoplastic transformation, cell growth inhibition, and enhanced apoptosis and anti-angiogenicity, through the inhibition of eicosanoid production from n-6 fatty acids; and suppression of cyclooxygenase 2 (COX-2), interleukin 1 (IL-1) and IL-6 gene expression by n-3 fatty acids. Recent intervention studies in breast cancer patients indicate that n-3 fatty acids, and docosahexaenoic acid (DHA) in particular, increase the response to chemopreventive agents. In patients with colorectal cancer, eicosapentaenoic acid (EPA) and DHA decrease cell proliferation, and modulate favourably the balance between colonic cell proliferation and apoptosis. These findings should serve as a strong incentive for the initiation of intervention trials that will test the effect of specific dietary patterns in the prevention and management of patients with cancer.

08 July 2004

Hundreds of Millions Exposed to Carcinogenic SV40 Virus

Vaccine scandal revives cancer fear
19:00 07 July 04 New Scientist

Many millions more people than previously thought might have been given polio vaccine contaminated with a monkey virus linked to cancer.

It has been known since 1960 that early doses of polio vaccine were widely contaminated with simian virus 40, or SV40, which infects macaque monkeys. Tens of millions of people in the US and an unknown number in other countries, including the UK, Australia and the former Soviet Union, may have been exposed prior to 1963.

The contamination occurred because the kidney cells the vaccine virus was grown in came from monkeys infected with SV40. Health officials say the problem was eliminated after 1963.

Now Michele Carbone of Loyola University Medical Center in Chicago has announced results that suggest the Soviet polio vaccine was contaminated after 1963, possibly until the early 1980s. "Is there infectious virus? The short answer is, yes," Carbone told the Vaccine Cell Substrate Conference 2004 in Rockville, Maryland, last week.

The vaccine was almost certainly used throughout the Soviet bloc and probably exported to China, Japan and several countries in Africa. That means hundreds of millions could have been exposed to SV40 after 1963.