30 June 2004

Pollen Update for Boston Area


When out running this morning, I noticed a mild pollen effect. Pollens are up into the medium range in Boston. As noted previously, a few seconds per minute of broadcasting the correct frequency with the Advanced Biophoton Analyzer (ABPA) will eliminate allergy symptoms.

label start
dwell 8
duty 50
pulse 64 75
converge 27 1
464452
pause 52
goto start

28 June 2004

Consumer Alert: Is Your Doc Paid by Drug Companies to Prescribe Your Medication?

Medication error is the fourth leading cause of death in the United States. A previous posting documented a recent study showing that about 25% of prescriptions written by physicians are for patients with conditions that are listed in bold print on the warning label on the drug package. In addition, drug companies are offering to pay physicians large sums of money to prescribe specific medications. Consumers must make their own safety a priority. They should always read the Physicians Desk Reference information for risks and contraindications on any drugs they are taking. They should also ask their physician if they or their institution have any financial interest in prescribing a specific drug before taking it.


As Doctors Write Prescriptions, Drug Companies Write Checks
Gardiner Harris, 27 June 2004

The check for $10,000 arrived in the mail unsolicited. The doctor who received it from the drug maker Schering-Plough said it was made out to him personally in exchange for an attached "consulting" agreement that required nothing other than his commitment to prescribe the company's medicines. Two other physicians said in separate interviews that they, too, received checks unbidden from Schering-Plough, one of the world's biggest drug companies.

"I threw mine away," said the first doctor, who spoke on the condition of anonymity because of concern about being drawn into a federal inquiry into the matter.

Those checks and others, some of them said to be for six-figure sums, are under investigation by federal prosecutors in Boston as part of a broad government crackdown on the drug industry's marketing tactics. Just about every big global drug company — including Johnson & Johnson, Wyeth and Bristol-Myers Squibb — has disclosed in securities filings that it has received a federal subpoena, and most are juggling subpoenas stemming from several investigations...

26 June 2004

Pollens: Orlando, Tampa, Boston


Recently, I found out that some people are eliminating allergy symptoms by creating a list of all the frequencies I have posted on this web site, so I'll try to post more often.

On Wednesday and Thursday I was in Orlando. Pollen count was low. Grass and ragweed was predominate. I began to experience mild asthma symptoms (a rare occurence for me) in the upper respiratory tract. So I put the following frequency in my Advanced Biophoton Integrator in Boston for broadcast to Orlando. It cleared it right up. I don't think this is grass or ragweed. It is likely to be a flowering plant in the Orlando area.

label start
dwell 13
duty 50
pulse 64 75
converge 25 1
477666
pause 47
goto start

I've found that broadcasting 7-13 seconds each minute will keep my system clear of pollens.

On Friday in Tampa, the pollen was different. Ragweed, grass, and chenopod were predominant. What I think is chenopod is causing tired and sleepy symptoms. The following frequency cleared it right up.

label start
dwell 8
duty 50
pulse 64 75
converge 15 1
476245
pause 52
goto start

This pollen is killed more easily and only requires 8 seconds per minute of transmission.

On returning to Boston on Saturday, the pollen count is headed up from grass and sorrel/dock. Mild allergic symptoms appear to be sorrel. The following program takes care of it:

label start
dwell 8
duty 50
pulse 64 75
converge 11 1
477767
pause 52
goto start

For those not familiar with the mechanism of allergies, the pollens are growing pathogens in your body. If your immune system can knock them out quickly, you do not have allergic symptoms. The allergic symptoms are your body's failed attempt at killing them. In the case above, a few seconds of ABPA transmission every minute will mimic the functioning of an excellent immune system.

This is one of the best demonstrations of the effectiveness of electronic medicine. Give me a common allergic symptom and I will knock it out in 2 minutes 100% of the time. You don't need a clinical trial to prove effectiveness when effectiveness is 100%.

Furthermore, with the ABPA you can keep the allergic symptoms knocked out while exercising as I have shown in previous postings. And when you turn the ABPA off, the allergic symptoms immediately come back, so can also easily demonstrate that Koch's postulates are met for this process.

Click here for information on how to use an ABPA for this purpose.

20 June 2004

Life Extension: Longevity Gene Activated by Diet Restriction



'Longevity' gene, diet linked SIRT1 may play role in why calorie restriction leads to longer life in rats
By David Secko, The Scientist, 18 Jun 2004

Experiments on yeast, worms, and mammals have all revealed that cutting calories extends lifespan, but how is this occurs is largely unknown. Now, two independent studies, one reported in the June 18 Science, from David Sinclair's laboratory at Harvard Medical School, and the other reported in the June 3 Nature, from Leonard Guarente's laboratory at Massachusetts Institute of Technology, have uncovered molecular mechanisms linking aging, diet, and a gene called SIRT1.

"People have been studying calorie restriction for 70 years, yet the mechanism is not known," said Sinclair, adding that this new work has finally started to suggest a primary mechanism...

In the Science study, Sinclair, lead author Haim Cohen, and their colleagues investigated how SIRT1 was involved in calorie restriction in rats, keying on the concept that aging results in cell loss over time. "Caloric restriction and genetic manipulations that extend lifespan typically protect cells from death," Sinclair told The Scientist.

Sinclair and colleagues found the SIRT1 expression was higher in 12-month-old rats fed a calorie-restricted diet. This finding also extended to in vitro cell culture, with serum from the calorie-restricted rats inducing SIRT1 expression. The addition of insulin and insulin-like growth factor 1 (two factors known to be involved in extending lifespan) to the calorie-restricted serum blocked the induction of SIRT1.

Further investigation revealed that SIRT1 acted on a DNA repair factor Ku70, which was then capable of repressing the apoptosis-inducing protein Bax. This suggested a mechanism in which SIRT1 protected cells from apoptosis.

"We propose that one-way calorie restriction extends lifespan is by increasing SIRT1 expression, thereby promoting the long-term survival of irreplaceable cells," said Sinclair.

Update on April 2004 Flu Alert: Lingering Cough



Several people have lingering coughs from the April 2004 flu. These frequencies may clear it up:

repeat 120
dwell 60
duty 50
pulse 64 75
converge 27 1
466445 344455 254335 166635 #parasite
converge 57 1
366565 #virus
end repeat

---------------------

In April 2004, I worked with people in California, Connecticut, and Massachusetts on an upper respiratory infection that appears to be a corona virus transmitted by a microscopic parasite (as was SARS). The parasite grows in the head and respiratory tract and keeps emitting the virus as the immune system trys to kill it.

It can easily go to bronchitis and is responsive in the early stages to Oscillicocinum 200C. It creates brain fog and can lead to forgetfullness, falling, and poor decisions.

The frequencies below will eliminate most of the symptoms and should stop all of them if caught early.

label start
dwell 60
duty 50
pulse 64 75
#begin parasite
converge 25 1
116545
converge 13 1
116080
244195
355110
355150
355500
355510
355760
355890
356000
455565
#end parasite
258643
165345
265766
106475
245662
245356
36566
257466
237257
165545
36454
35645
75145
244245
128664
175622
converge 16 1
645764
864257
#cleanup
converge 36 1
246654
converge 46 1
254514
253476
goto start

Click here for comments and plasma device frequencies

Wired News: Government Slowly Getting Serious About Medical Error


Dragging Doctors to the Info Age
By Randy Dotinga, Wired News: 02:00 AM Jun. 18, 2004 PT

Frustrated by the hospital industry's achingly slow adoption of basic technological safeguards, the Bush administration and Congress are gearing up to put more pressure on doctors and administrators. For perhaps a decade or more, however, millions of patients will continue to endure the high risk of deadly medical mistakes because there's no computer to catch the errors.

U.S. hospitals are hardly as computerized as, say, the Department of Motor Vehicles or the Internal Revenue Service. Physicians can spend entire days without touching a keyboard, and nurses routinely track patients' progress through a series of handwritten notes passed from person to person.

"It's this huge, ridiculous game of telephone," said surgeon Dr. Robert Wachter, co-author of a new book exploring why medical errors kill tens of thousands of Americans each year.

More potential disaster looms on the medication front, where the "chicken scratch" on prescription forms often confuses pharmacists, who end up providing the wrong drugs and, in some cases, killing people.

Enter the politicians -- finally. On the federal level, members of Congress announced plans earlier this month to form a caucus to push for better hospital technology. The Bush administration is drawing praise for hiring a health information technology czar, while former House Speaker Newt Gingrich has formed a think tank devoted to exploring the challenge of equipping hospitals.

"The government has finally realized in the last month or two that unless it steps in ... many people will die because of the absence of reasonable computer systems in American health care," said Wachter, professor of medicine at the University of California at San Francisco and co-author of Internal Bleeding: The Truth Behind America's Terrifying Epidemic of Medical Mistakes.

Cancer: A Matter of Cellular Programming


Maher, Brendan A. (2004) The Makings of a Microarray Prognosis. The Scientist, March 15, pp. 32-36.

"Countless things can go wrong in the complicated cell division process. Checkpoints fail, genomic instability increases, and when anarchy reigns, cancers spread. In trying to assess what is doing the damage and predict the damage yet to be done, doctors have an admittedly blunt set of tools to porfile renegade cells. Histopathology andgeneral prognostic indicators such as health, age, and metastatic spread do little to account for the breadth of tumor variety even within fairly specialized tumor types.

"Snce researchers first stated toying with microarrays, they have considered the molecular pofiling of tumors to be an important potential use. The genes that switch on and off may reveal a cancer's biology and future, and, some hope, its responses to a variety of treatments."

Metastatic models of cancer are changing due to analysis of gene behavior in tumors. The ability of a tumor to spread to other organ systems was previously considered a phenomenon that occurred late in the tumor life cycle. It now appears that certain tumors are prone to metastasis from the beginning and that they have subpopulations of cells with affinity to certain organ systems.

The implications of this is that treatment should be different for cells prone to metastasis and those with less aggressive tumors should not be exposed to unnecessary chemotherapy.

For example, a recent study looked at 20 year old tissue samples from breast cancer patients without lymph node metastasis. At that time they were not treated with chemotherapy. Out of 98 women younger than 55, there were 34 with a recurrence. Today, in the U.S., 95% of these patients would be treated with chemotherapy. This means that 65% of these patients would not need chemotherapy, but would get it anyway leading to excess costs and morbidity.

So it looks like patients should demand the health care system do a more detailed examination of their tumor before they rush to chemotherapy. It could not only save them severe debilitating effects, it could reduce healthcare costs.

08 June 2004



Calorie restriction molecular mechanism found
Life Extension Update, 3 June 2004

Research in mouse tissue conducted by MIT professor of biology Leonard Guarente and colleagues, published in the online version of Nature (http://www.nature.com/nature/) on June 2 2004, found that calorie restriction effects a protein that controls whether fat is stored or released. Calorie restriction has been found to extend the lifespan of every species in which it has been tested, and many humans have adopted the diet in hope of extending their own lives. The protein is the product of the mammalian gene sirtuin 1 (Sirt1), similar to the SIR2 gene that has been found to mediate the effects of calorie restriction in yeast. When Sirt1 senses short-term famine, such as is mimicked by calorie restriction, it turns off the receptors that keep fat in fat cells, and fat is released or metabolized rather than stored. The authors explain that the “Sirt1 protein activates a critical component of calorie restriction in mammals; that is, fat mobilization in white adipocytes. Upon food withdrawal the Sirt1 protein binds to and represses the genes that are controlled by PPAR-gamma, the fat regulator.”

Dr Guarante elaborated, “The ability of fat cells to sense famine and release the fat is regulated by this gene. We like to think this applies to people as well as mice, but we don’t know for sure. If we could make this happen in people, it wouldn’t just make them live longer; it might also help prevent diseases of aging, like cancer, diabetes and heart disease . . . If we could make a drug that would bind to Sirt1 and fool the body into thinking that it needed to release that fat, then maybe people could get the benefits of calorie restriction without the side effects.”

Because white adipose tissue makes hormones, including leptin which controls satiety, Guarente speculates that fat cells also tell the body how fast to age. He added, “Conversely, fewer fat cells tell the body that it’s time to hunker down for survival. This means that evolutionarily speaking, fat plays a very important role.

07 June 2004

Longevinex Gene Activation Supports Life Extension


Longevinex™: First Red Wine Dietary Supplement To Confirm Biological Activity (Activation of Sirtuin 1 Gene)

An independent laboratory test confirms that Longevinex™, a red wine dietary supplement, exhibits significant biological activity, potently activating the Sirtuin 1 gene, known as a survival factor in living cells. Resveratrol is a component of red wine and is well known for its healthy properties.

Among 14 brands of resveratrol dietary supplements independently tested, only Longevinex exhibited significant biological activity. At equal concentrations, none of the other 13 brands of resveratrol exhibited significant Sirtuin 1 activity. Longevinex (3.551) has six times greater effect than the best other resveratrol supplement (0.499). A number of resveratrol supplements exhibited biological activity that was barely measurable.

Surprisingly, three non-resveratrol supplements (IP6 rice bran extract 0.966, butyric acid 1.214 and genistein soy extract 1.393), produced 2-3 times greater Sirtuin 1 gene activity than 13 brands of resveratrol, though these products were not considered to exhibit significant biological activity either.

A freshly published report in Nature Magazine last week confirms that resveratrol mimics the beneficial biological effects of calorie restriction. Previous studies have shown that pure resveratrol exhibits the strongest Sirtuin 1 activation levels among 17 polyphenol molecules tested. [Nature 425(6954):191-6, Sept 11, 2003].

But the question remains, do dietary supplements exhibit significant biological activity similar to the laboratory-grade resveratrol? This study confirms that it is possible for resveratrol dietary supplements to exhibit significant biological activity (i.e. Sirtuin 1 gene activation) at achievable concentrations in living cells.